PE cardiac

dextrocardia

condition of the heart on the right side, either rotated, displaced or mirror image

apical impulse usually felt

fifth left intercostal space at mid clavicular line

what side is more forward in the chest

right side

what happens during systole

the ventricles contact; ejecting blood from left of heart through aortic valve to aorta and right side of heart through pulmonic valve to pulmonary artery

what happens during diastole

the ventricles dilate; draws blood into ventricles as atria contract

S1

the first heart sound "Lubb"; as systole begins ventricular pressure rises and forces mitral(bicuspid) and tricuspid valve closed, preventing back flow.

S2

the second heart sound "dubb"; pressure in ventricles fall below aortic and pulmonary artery and the aortic and pulmonary valves close.
The end of systole

SPLIT S2

A2 is produced by the aortic valve closing, P2 by the pulmonic valve closing. A2 will be before P2 because the pressure in the right side of the heart is slightly less than the left so it takes longer.
Heard best during inspiration

S3

the filling of the ventricle, not a normal sound
Gallop, right after S2

S4

when atria contracts ensuring ejection; a stiff wall
increased resistance to filling because of loss of compliance of the ventricular walls; or increased stroke volume of high out-put states
Right before S1

When you have a HR of 68-72 ventricular systole is _______ than diastole.

ventricular systole is shorter than diastole

When your HR increases what happens to the length of systole and diastole.

the two phases of the cardiac cycle tend to approximate each other in length

The heart lies more _________ in infants and young children

horizontally

During pregnancy the hear is shifted how

shifted toward a horizontal position and there is a slight axis rotation

A through examination of the heart requires a patient to assume what positions

Sitting erect
leaning forward
lying supine
Left lateral recumbent positions

Apical impulse can be made visible by having the patient

sit up, bringing the heart closer to the anterior wall

What would an apical impulse indicate that is more widely distributed, fills systole or displaced laterally and downward

increased cardiac output
Left ventricular hypertrophy

A lift along the left sternal border may indicate

Right Ventricular hypertrophy

Displacement of apical impulse to right without a loss or gain in thrust suggest

dextrocardia
diaphragmatic hernia
distended stomach or
pulmonary abnormality

What is a thrill

Fine palpable rushing vibration, palpable murmur grade IV or greater.
Generally indicates turbulence or disruption of expected blood flow, due to defect of closer of one of the valves

Thrill in systole felt at suprasternal notch &/or second and third right intercostal space most likely

Aortic Stenosis

Thrill in systole at suprasternal notch &/or second and third left intercostal space most likely

Pulmonic Stenosis

Thrill in systole at fourth left intercostal space most likely

Ventricular septal defect

Thrill in systole at Apex most likely

Mitral regurgitation

Thrill in systole at left lower sternal border most likely

Teatralogy of Fallow

Thrill in systole at left upper sternal border, often with extensive radiation

Patent ductus arteriousus

Thrill in diastole in right sternal border most likely

Aortic Regurgitation

Thrill in diastole at Apex most likely

Aneurysm of ascending aorta
Mitral Stenosis

Aortic Valve area

second right intercostal space at right sternal border

Pulmonic Valve area

Second left intercostal space at left sternal border

Erb's Point, Second Valve area

Third left intercostal space at left sternal border

Tricuspid valve area

fourth left intercostal space at left sternal border

Mitral valve area

Fifth left intercostal space at the midclavicular line

Where is a split 2 best heard

pulmonic auscultatory area

S1 best heard where

at the apex

S2 best heard at

aortic and pulmonic area

Splitting S2 is pathological when

it is fixed; unaffected by respiration
large atrial septal defects
ventricular septal deftect with R to L shunting
R ventricular failure

S3 can be heard better if

filling pressure is increased or ventricular compliance is reduces.
Heard best in left lateral position

Raising the legs does what

Increases Venous Return

An isometric grip does what

Increases arterial pressure, increases after load
increases mitral regurgitation

What is and when does paradoxic splitting of S2 occur?

Occurs when closure of aortic valve is delayed, so P2 occurs before A2.
Left Bundle Branch Block

A wide splitting of S2 is indicative of

Right Bundle branch block
Stenosis delays pulmonic valve closer
Pulmonary HTN delays ventricular emptying
Mitral regurgitation induces early closure of aortic valve

The two main causes of Heart Murmurs are:

1. Stenosis of a valve; harder to open, so hear when you normally do not.
2. Regurgitation: valves no longer fit together tightly and allow blood to flow backwards.

Other causes of Heart Murmurs

High output demands (anemia, pregnancy)
structural defects other than valve (hole in septum)
diminished strength of contraction of Heart
altered blood flow through major blood vessels near heart
Obstructive disease (atheroscleortic dz)

Mitral Stenosis PE findings

Heard best at apex, does not radiate
Diastolic rumble, more intense in early and late diastole.
Opening Snap following S2
Palpable thrill at apex in diastole common

Mitral Regurgitation PE findings

Heard best at apex, radiates to left axilla/ subscapular region
Holosystolic murmur, may obliterate S2
Prominant S3
displaced apical impulse

mitral valve prolapse PE findings

Heard best at apex & left lower sternal border
easily missed in supine position
Mid-systolic click, high pitched
Late systolic murmur may follow (MR)
Click will occur earlier while standing/ Valsalva (decreased afterload)
Click will occur later while squa

Aortic Stenosis PE findings

Heard over Aortic area, radiates along left sternal border and to carotid with palpable thrill
Mid-Systolic ejection murmur;
crescendo-decrescendo
murmur shifts depending of severity of disease

Aortic Regurgitation PE findings

Best patient seated bent forward, aortic area/ erb's point
Early Diastolic blowing murmur
Widened pulse pressure
low diastolic pressure
Water-Hammer (corrigan) pulse

Sub-aortic Stenosis PE findings

Fibrosis Ring 1-4 cm below aortic valve causes murmur
Murmur fills systole; crescendo-decresendo
thrill often palpable during systole at apex and right sternal border
jugular venous pulses prominant

Pulmonic Stenosis

Systolic ejection murmur
radiates to left and into neck
S4 common in R ventricular hypertrophy
thrill in second and third intercostal spaces

Pulmonic Regurgitation

Difficult to distinguish from aortic regurgitation
Echo definitive in high pressure, but less definitive in low pressure

Tricuspid Stenosis

Diastolic rumble along left sternal boarder
increases with inspiration
jugular venous pulse prominent especially a wave. slow fall of v wave

Tricuspid Regurgitation

Heard best at left lower sternum
Holosystolic murmur, over right ventricle
Increases with inspiration (increased volume in R ventricle with negative pressure)
Systolic c-V wave in jugular venous pulsations

hypertrophic cardiomyopathy

Valsalva; increases (decreases venous return, decrease preload)
Squatting to standing; increases
standing to squatting; decreases
Leg elevation; decreases (increased venous return, increases preload)

A murmur from what type of shunt can be increased by hepatic reflex

Right to left shunt; due to increasing right atrial pressure

A murmur from what type of shunt will be decreased by hepatic reflex

Left to Right shunt; due to increasing right atrial pressure will disappear brefiely

Patent ductus arteriousus may be associate with what type of murmurs

murmur extends past S2 and occupy diastole and machine like quality
disappear in first 2-3 days of life when closes

sinus arrhythmia

physilogic during childhood.
Heart rate varies in cyclic pattern usually faster on inspiration and slower on expiration.

Normal newborn heart rate

120- 170 bpm

Normal 1 year old heart rate

80 -160 bpm

Normal 3 year old heart rate

80-120 bpm

Normal 6 year old heart rate

75 - 115 bpm

Normal 10 year old heart rate

70-110 bpm

What abnormal heart sound is more common in older adults?

S4, decreased left ventricular compliance

Bacterial Endocarditis

bacterial infection of the endothelial layer of the heart and valves.
S/S: Fever, fatigue, murmur, sudden onset of congestive heart failure.
PE: neurologic dysfuntion, janeway lessions; painful small erythematous or hemorrhagic macules on palms and soles

Congestive Heart Failure

Heart fails to propel blood forward with its usual force, resulting in congestion in pulmonary or systemic circulation.
Right or
Left sided systolic HF; narrow pulse pressure
Left sided diastolic HF; wide pulse pressure

Most helpful clinical finding in Left sided heart failure

Jugular venous distension

Pericarditis

Sudden inflammation of the pericardium
S/S; sharp stabbing chest pain, movement/inspiration aggravate pain, pain relieved by leaning forward
PE; friction rub on ascultation

Cardiac Tamponade

excessive accumulation of effused fluids or blood between the pericardium.
S/S; chest pain relieved by sitting upright and leaning forward, difficultly breathing, snycope, pale/gray or blue skin, palpitations, rapid breathing
PE;
Becks triad: 1. jugular v

Cor Pulmonale

Enlargement of right ventricle secondary to pulmonary malfunction.
Chronically caused by COPD
Acutly by acute respiratory distress syndrome
S/S; fatigue, tachypena, exertional dyspnea, cough, hemoptysis
PE; pulmonary disease, wheezes & crackles on auscult

Sick Sinus Syndrome

Arrhythmias caused by a malfunction of sinus node.

Tetralogy of Fallot

Four abnormalities:
1. stenotic pulmonary valve
2. ventricular septal defect
3. overriding aortic valve
4. hypertrophy of right ventricle
S/S; dyspnea with feeding, poor growth, exercise intolerance, paroxysysmal dyspnea with loss of consciousness & centr

Ventricular Septal Defect

opening between the left and right ventricles; many small defects close spontaneously during first 2 years of life
s/s: recurrent respiratory infections, large rapid breathing, poor growth and symptoms of CHF
PE; holosystolic murmur heard best along left

Atrial Septal Defect

congential defect int he septum dividing the left and right atrium.
S/S; often asymptomatic, heart failure rarely in children,but can occur in adults.
PE; systolic thrill, systolic ejection murmur over pulmonic area, may have breif rumbling early diastoli

Patent Ductus Arteriosus

failure of the ductus arteriosus to close after birth.
S/S; small shunt can be asymptomatic and larger one dyspnea on exertion.
PE; dilated pulsatile neck vessels, wide pulse pressure, harsh, loud continuous murmur heard at 1-3 intercostal space with mach

Kawaski Disease

Condition causing inflammation in walls of small and medium-sized arteries throughout the body including coronary arteries.
S/S; fever, lasting longer than 5 days, conjunctivitis, strawberry tongue, cervical lymphadenopathy, erythemia of palms and soles,

Pre-eclampsia

Hypertension after 20th week of pregnancy and presence of proteinurea
> 160 mmHg systolic, >110 mmHg diastolic

eclampsia

Hyptertension after the 20th week of pregnancy and presence of proteinurea with seizures with no other identifiable cause
> 160 mmHg systolic, >110 mmHg diastolic

Raynaud Phenomenon

exaggeration of digital arterioles usually to cold exposure.
Most common in young healthy women
s/s: area feels cold/achy white and blue with cold exposure and red with reprofusion

Secondary raynaud phenomenon

associated with underlying connective tissue disorder, scleroderma or systemic lupus.
PE; ulcers on tips of digits, can appear smooth shiny & tight from loss of subcutaneous tissue

Temperal arteritis (Giant Cell arteritis)

Inflammatory disease of the branches of the aortic arch including temporal arteries.
Can lead to ischemia of supplied structures (masseter, tongue or optic nerve).
S/S; flue like (low grade fever, malasie, anorexia), polymyalgia of hips/ shoulders, headac

List the measurements for edema

1+: slight pitting (2mm), no visible distortion disappears rapidly
2+:Somewhat deeper than 1 (4mm), no readily detectable distortion, disappears 10-15 sec
3+:notably deep pit (6mm) last longer than 1 min, dependent extremity looks fuller and swollen
4+: v

JVP distension

Normal value >9 cm
If cant see its under 5 cm
Calculate the number and add 5 cm

hepato-jugular reflex

exaggerated when right heart failure is present
apply pressure to liver, increase in JVP with pressure, decreases when released

Watter Hammer (Corrigan) Pulse

greater amplitude than expected, a rapid rise to a narrow sumit and sudden descent.
Patent ductus arteriosus
aortic regurgitation

Paradoxic Pulse

exaggerated decrease >10mmHg in amplitude of pulsation during inspiration and increased amplitude during expiration.
Premature cardiac contraction
Tracheobronchial obstruction
bronchial asthma
emphysema
pericardial effusion
constrictive pericarditis

Pulsus bisferiens

two main peaks.
Aortic stenosis combined with aortic insufficency

pulsus alterans

Alteration of small and large amplitude
Left ventricular failure; more significant if pulse is slow

Jugular veins reflect what

the right side of the heart and offer clues to its compensation.
Level of jugular venous pulse indicates Right Atrial pressure.

Jugular A wave

First most prominent wave, result of a brief back flow of blood to vena cava during Right Atrial contraction.

Jugular C wave

transmitted impulse from vigorus backward push produced by closer of the tricuspid valve during ventricular systole.

Jugular V wave

Increased volume and concomitant increased pressure in right atria, after c wave late in ventricular systole

Jugular X slope

after C wave, caused by passive atrial filling

Jugular Y slope

After v wave, open tricuspid valve and rapid ventricular filling