Substance Abuse- Coun. 501

self-report inventories

instruments used for diagnostic purposes, they can be useful, but are not meant to be used in isolation from other assessment data

components of a psychosocial history

AOD use history, family history, social history, legal history, educational history, occupational history, medical history, and psychological and behavioral problems

referral programs

refer to a program that conducts objective assessments, has proper licensing, credentialed counselors

misuse

when a person experiences negative consequences from the use of AOD

substance abuse

the continued use of alcohol or other drugs in spite of negative consequences

addiction or dependence

is the compulsive use of AOD relgardless of the consequences

Early Full Remission

if the client has been in recovery for 1 to 12 months and none of the criteria for dependence or abuse are met

Early Partial Remission

if one or more criteria are met but the client cannot be diagnosed as substance dependence- does not meet three or more of the criteria

Sustained Full Remission

period of recovery is 12 months or longer without slips

Sustained Partial Remission

period of recovery is 12 months or longer but had slips but not full-blown relapse

On Agonist Therapy

client has a low probability of use due to external circumstances

In a Controlled Environment

if client is in prison or a supervised enviroment without AOD

Minnesota Model

4 components- 1- belief that client can change attitutude, 2- adheres to the disease concept of addiction, 3- long-term treatment goals- personal growth, 4- principals of AA and NA in treatment

Aversive conditioning techniques

using unpleasant stimuli to reduce desire - nause, apnea, electric shock

Heroin withdrawal medications

methadone, suboxone

treatment modalities

individual, group, and family counseling, support groups, lifestyle changes, education and aftercare

treatment settings

inpatient and residential settings, partial hospitalization, day treatment, intensive outpatient, and outpatient treatment

Drug Abuse Prevention and Control Act

was passed by congress in 1970. As part of this law, drugs are placed in one of five shcuedules with requirements assoicaited with each schedule.

Schedule I drugs

have a high potential for abuse- they have no currently accepted medical use in treatment in the US and a lack of a safe level of use under medical supervision- heroine, quaalude, LSD and marijuana

Schedule II drugs

high abuse potential and can lead to psychological or physical dependence- can be used in medical treatment- morphine, PCP, cocaine and methamphetamine

Schedule IV drugs

Valium, Xanax- related to public policy- can have same abuse potential

Benzodiazepines

most widely prescribed drugs, valium, xanax, ativan, klonopin, serax, librium sometimes used for the treatment of panic attacks

Benzos

most widely abused, fat soluable, 1/2 life, xanax and valuim- most widely abused

Barbituates

prescribed for sleep, used for seizure control- phenobarbital, seconal, quaaludes

Opiods

prescribed as an alangesic- pain relief, vicadan olycontin, percocet methadone- could cause respiritory depression

alchohol

most commonly used intoxicant- affect GABA/inhibition of stimulation, blocks panic and anxiety

Beer

3%-6% alcohol

Wine

11%-18% alcohol

Liquor

25% or more alcohol

CNC- central nervous system depressants

sedates, relaxes and calms- downers

Nicotine

causes more deaths than alcohol, most widely used

Cocaine

most addictive, smokeable form crack- rapid high intense crash, high 20 minutes to an hour

swallowing pills

takes 15-30 minutes to get absorbed into the bloodstream

snorting

takes 5-7 minutes to produce a high

IV use

takes 15-30 seconds to produce a high

smoking

takes 8 seconds to produce a high

Methamphetamine

speed, crank, meth, crystal- can be snorted or smoked, synthetic, high up to 4 hours, pschomotor stimulation, allertness, mood elevation, withdrawal is not medically dangerous, but psychologically devastating (cocaine too)

Hallucinogens

escape reality, LSD, mushrooms, morning glory, marijuana- can cause panic reactions, injuries to self/others, flashbacks- could last for years

Marijuana

most widley used illegal drug, active ingredient THC, gateway drug, amotivational drug- "Careless drug

alchohol overdose

common- being drunk- staggering, slurred speech, extreme disinhibition, blackouts, stomach spasms- vomit, rapid ingestion may result in coma or death

overdose

depress the central nervous system- 10-15 times the therapeutic dosage- barbituates often used in suicides

accidental overdose

combination of alcohol and benzos- xanax, ativan

inhalants

produces euphoria- nitrous oxide, poppers, white out, could result in hallucinations, brain paralysis, or coma

anabolic steroids

used to improve athletic perfromance- muscle mass- infertility, baldness, aggression

club drugs

ecstasy- MDMA, special K, pohypnol- date rape drug, GHB- diminishes inhibitions- enhances sexual experiences

central nervous system depressants

alcohol, minor tranquilizers

central nervous system stimulants

cocaine, mehtanphetamine

Impaired Model

no addicts are this way by nature- prognosis: death due to use

Dry Moral Model

maintain complete abstinence or it's a sin- based only on willpower, uses guilt and remorse, religious conversion

Wet Moral Model

advocates for controlled drinking, supports self-will, goal- drink responsibly

Psychoanalytic Model

use is due to an underlying personality disorder, most expensive and most popular

Family Interaction Model

focus on family- successful with intervention, pronosis: poor without full family involvement

AA Model

addiction is defined as a physical allergy and mental obsession, has a support system, prognosis: fair to good

Love Model

Addict doesn't perceive themselves as sufficiently loved

Substitution Model

created by an inability to cope with anxiety due to absitinence, treat with substitute medications that could be addictive, but not the person's drug of choice, pronosis is poor

Biopsychosocial Model

looks at a variety of factors in explaining the addiction i.e. biological, psychological, environmental, and cognitive

Social Learning Model

use is the result of the environment

Old Medical Model

self-induced deterioration due to drinking, treatment- detox. and meds, treat the symptoms and let them go, implies one can drink if no medical problems exist

Disease Model

E.M. Jellninek - physiological susceptibility to disease exacerbated with continued use, requires abstinence + education, does not require psychological help, unless dual diagnosis

Dopamine

pleasure seeking transmitter

Norepinephrine

adrenaline, energy transmitter

Serotonin

mood regulator transmitter

GABA

inhibitory (Stops inhibitions) transmitter

Glutamate

It is always excitatory (GO), usually due to simple receptors that increase the flow of positive ions

Acetylcholine

plays a role in skeletal muscle movement, muscle control

Natural Opiates

andogenous opiates- endorphins help pain relief made from poppy

Opiods

man-made synthetic opiate, any synthetic narcotic that has opiate-like activities but is not derived from opium

Brain effect

Brain gets tricked with drug abuse b/c the neurotransmitters are confused with the natural neurotransmitters and are passed through the blood-brain barrier

electrical impulse

gets stimulated through drug use

Homeostasis

balances the brain- tries to find balance by shutting down or opening up more receptor sites

tolerance

as receptors are closed, a person can never get that "First High", needs to take more to get more of a high

Limbic System

pleasure and survival part of the brain

cerebral cortex

learning, judgment part of the brain

VTA Ventral Tagmental Area

rewards circuit of the brain, where dopamine is produced

Amygdala

stores emotional memories and pleasurable memories

Hippocampus

intellectual memories, learning (marijuana affects this area, pot-head can't remember)

Dopamine Depletion Hypothesis

addiction begins at this stage b/c the brain stops producing the receptors, abstinence must occur in order for the brain to start producing receptors again

reuptake

recycled, the reabsorption of a secreted substance by the cell that originally produced and secreted it, for example, affects serotonin

SSRI selective seratonine reuptake inhibitor

are a class of compounds typically used as antidepressants in the treatment of depression, anxiety disorders, and some personality disorders.

Alcohol disease

over time mitochondria becomes misshapened - first sign of addiction

mitochondria

parts of the cell that produce energy

esophagitis

drying of the esophagus

Liver disease

enzomatic breakdown- enzymes overactive,

hepatitis

fatty liver, liver enlarged

serosis

scarred liver, can cause cancer

bial

accumulated in body jaundice

THIQS tetrahydroisoquinenlines enzyme

enzyme that gets produced in the brain of an alcoholic, stimulates opiate receptor sites- produces euphoria

Meds to stop drinking

NTA, antobuse, naltrexone revia- used for relapse patients

naloxo

used to stop withdrawal

opiate antagonist

blocks the opiate

compral accopracy

drug taken after acute withdrawal- helps with cravings

compliance

client needs to take medication as directed

naltrexone

is an opioid receptor antagonist used primarily in the management of alcohol dependence and opioid dependence

Functional Alcoholic

more at risk for chronic medical problems than an alcoholic

cross tolerance

addicted to one substance, tolerance is higher for other meds. in that category

cross addiction

addicted to more than one category

poly-substance dependence

abuse dependences- 3 or more categories of addiction i.e. huffing, smoking pot, and alcohol abuse

rebound effect

opposite of intoxicating effect i.e. drink a lot- sleep, blood alcohol level drops, person wakes up needs more alcohol

cocaine - high

crash withdrawal could lead to substance abuse psychosis or suicide

synergistic effect

combining 2 drugs for a better high i.e. alcohol + a Benzo= multiplied effect, can cause accidental deaths

Acute withdrawal

lasts 1-5 days

Benzo withdrawal

lasts 4-14 days, need medically managed care

Post Acute Withdrawal or Protracted Withdrawal

more psychological dependence- could take up to 2 years- inability to solve simple problems

PAW symptoms - six major types

1-an inabiltiy to think clearly 2-memory problems 3-emotional overreaction/numbness 4-sleep problems 5-physical coordination problems 6- stress sensitivity

regenerative PAW

symptoms gradually improve the longer a person stays sober, brain rapidly returns to normal

degenerative PAW

symptoms get worse the longer a person stays sober, relapse prone, person may collapse physically or emotionally- possible suicide

stable PAW

symptoms essentially remain the same, frustrating b/c people believe they should get better with sobriety, but need to learn to manage these symptoms

intermittent PAW

symptoms come and go, may have improvement then episodes that are severe, may go away, may occur periodically

Managing PAW

Stabilization, Education and Retraining, Self Protective Behavior, Nutrition, Exercise, Relaxation

CIWA- clinical institute withdrawal assessment for alcohol

scaled assessment used to dertermine Post Acute Withdrawal

Predictor of adolescent substance abuse

verbal abuse, decrease social involvement, absenteeism, truancy, stealing, involvement with peers who use