patho

Disorders of the kidneys

developmental defects
infections
altered immune responses
neoplasms

dysgensis

failure of an organ to develop normally

agenesis

complete failure of organ to develop

hypoplasia

failure of an organ to reach normal size

potter syndrome

characteristic facial features of newsborns with renal agensis

cystic renal dysplasia

microscopic features, is due to renal parenchymal malfomation of the fetal kidney

obstructive uropathy

a condition in which the flow of urine is blocked, causing it to back up and injure one or both kidneys

autosomal-recessive polycystic disease

small elongated cysts that form in the collecting ducts and maintain contact with the nepron of orgin

autosomal-dominat polycystic kidney disease

adult kidney disease-formation of destructive fluid filled cysts in the kidney and other organs

cystic disease of the kidney

fliud filled sacs or segments of a dilated nepron

kidney stones

crystaline structures that form from components of the urine

magnesuim ammonium phosphate stones

struvite stones

urine acid stones

formed in acid urine with ph of approximately 5.5, gout

cyctinuria

inherited disorder of amino metabolism

uretoroscopic removal

passage of cath through urethra into bladder and ureter

percutaneous removal

insertion of needle through flank into collecting system than neproscopre is inserted

extracorporeal lithotripsy

acoustic shock waves fragment calcull onto sandlike particles and pass in urine

pyelonephritis

characterized by renal inflammation and fibrosi induced by recurrent or persistant or persistant renal infection

medullary cystic disease

an inherited condition in which cysts in the center of each kidney causes the kidney to gradually lose thier ability to work

azotemia

presence of nitrogenous waste int he blood

oligurea

low output in the urine

glomerulonephritis

disease that provoke a profilerative responce of the endotheial, mesangeal, or epithieal cells of the glomeruli

Acute proliferative glomerulonephritis

resent onset of hematuria and proteinuria, impairment of kidney function (atoxemia), and salt and water retention causing edema and hypertension

rapidly progressive glomerulonephritis

progression of renal failure over days to weeks,in most case in context of nephritic presentation, typically associated with pathologicial finding of extensive glomerular cresent formation

nephrotic syndrome

kidneys are damaged causing them to have protienurea, hyperlipidemia, hypoabumineria, and lipiduria

membranous glomerulonephritis

caused by the thicking of part of the glomerular basement membrane

minimal change disease-lipid nephrosis

can lead to nephrotic syndrome

focal segmental glomerulosclerosis

scar tissue that forms in parts of the kidney call glomeruli

Iga nephropathy

antibodies called Iga build up in the kidney tissues. it also called bergers disease

acute tubular necrosis

damage to the tubule cells of the kidneys

chronic glomerulonephritis

advanced stage of a group of kidney disorders, resulting in inflammation and gradual, progression destruction of the glomeruli (internal kidney structures)

renal tubular acidosis

occurs when the kidneys fail to excrete acids into the urine

wilms tumor

kidney cancer that occurs in children

renal cell carcinoma

kidney cancer that starts in the lining of very small tubes (tubules) in the kidney