GU Phys and Path - Body Fluid Compartments

Kidneys

balance water intake with water excretionbalance electrolyte intake with electrolyte excretion

Insensible Water Output

evaporation from respiratory tract and from skin (700 ml/day, 300-400 ml/day)

Sweat Water Output

highly variable, usually about 100ml1-2L/Hr during exercise

Feces Water Output

normally 100 ml/day or less

Cellular Membrane

separates ICF from ECF

Capillary Membrane

separates plasma and interstitial fluid

ICF

fluid/cytoplasm inside the 750-100 trillion cells of the body40% of total body weight in malein RBCs

Plasma

non-cellular portion of bloodhas higher concentration of proteins than interstitial fluid

Protein

does not pass through, stays in blood capillaryBuffer of ICF

Polycythemia

High RBCs

Anemia

Low RBCs

Transcellular Fluid

Joint fluid, CSF, GI Fluids, makes up 1L of total body water

Passive Transport

DOWN Concentration gradientDIFFUSION (simple or facilitated)

Active Transport

needs energy, whether by ATP or otherwise

Primary Active Transport

needs ATP

Secondary Active Transport

Not via ATP

Co-Transport

Symport

Counter-Transport

Antiport

Osmosis

concentration gradient of water# of particles matters most

Hydrostatic Pressure

water pushing on watercauses fluid to leave the plasma

Osmotic Pressure (Oncotic)

water pulling on water, tonicitycell vs solutionpulls fluid into the plasma

Isosmotic

2 solutions with the same osmolatiry

Hyperosmotic

solution with > osmolarity than the reference solution

Hypo-osmotic

a solution with < osmolarity than the reference

When to Use Hypertonic

increased intracranial pressureuse Saline

Hypovolemic Shock

hemorrhage triggers release of vasopressin from Posterior Pituitaey Glandwidespread vasoconstriction

Extracellular

more Na+ and Cl-in plasma

Intracellular

2/3 of body water

Mechanisms Manage Salt Levels

1. Hypothalamus and ADH2. Adrenal Cortex and Aldosterone

Sodium

primary cation of extracellular Fluid

Potassium

Primary cation of intracellular fluid

Bicarbonate

primary buffer of plasma

Hypernatremia

blood levels of sodium are too highrelease of ADH to stimulate thirstdistal convoluted tubule opens aquaporins so water can be reabsorbed and dilute sodium

ADH

opens aquaporins for water reabsorption

Hyponatremia

excess water or loss of Na+will cause cell swellingdecrease in ADH from pituitaryDistal Convoluted Tubule used to reabsorb Na and excrete K

Baroreceptor Reflex

second by second management of BP

Decreased BP

CN IX to brainstemsympathetic stimulaitonincreased CO Vasconstriction

Inxcreased BP

CN IX to brainstemCN X to parasympatheticdecrease CO

Juxtaglomerular Cells

make and release renin when BP drops

Filtration

movement of water and solutes from capillary into the interstitial spacedriven by hydrostratic pressure

Reabsorption

movement of water and solutes back into blood from interstitial spacedriven by oncotic pressure

Renin

in Liver, converts angiotensinogen to angiotensin I

Angiotensin I

goes to lung for ACE

ACE

angiotensin-converting enzyme, from lungs converts angiotensin I to angiotensin II

Causes of Edema

1. Increase capillary hydrostatic pressure2. decrease plasma proteins3. Increase capillary permeability4. Blockage of lymph return

Kwashiorkor

Malnutrition produced by a severely inadequate amount of protein in the diet.

Pre-eclampsia

When a pregnant woman develops high blood pressure and protein in the urine after the 20th week (late 2nd or 3rd trimester) of pregnancy.

Angiotensin II

goes to adrenals, Hypothalamus, and the Sympathetic NS