Alpha-Adrenergic Drugs
(NE)
stimulate alpha-1 receptors to contract smooth muscle
Alpha-Adrenergic Blocking Drugs
stop the synthesis, storage, release or reuptake of NE
Beta-1 Adrenergic Drugs
(EPI)
stimulates the heart
Beta-2 Adrenergic Drugs
(EPI)
brochodilation, decreases uterine contraction, and relaxes smooth muscle
Beta-1 Adrenergic Blocking Drugs
antagonize the beta effects of EPI and NE on the heart
Beta-2 Adrenergic Blocking Drugs
antagonize the beta effects of EPI and NE on the bronchi, blood
vessels, and uterus
Cholinergic Agonists
enhance signaling of ACh
Cholinergic Antagonists
decrease the parasympathetic nervous systems function
Non-depolarizing (competitive) Neuromuscular Blockers
blocks neuromuscular receptors preventing the depolarization and
contraction of a muscle cell
Depolarizing Neuromuscular Drugs
depolarize cells and then is not degraded by AChE